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Background:
Posterior circulation stroke may rarely be associated with occlusive disease in the
anterior circulation, such as in the context of a direct (fetal) origin of the posterior
cerebral artery (PCA) from the internal carotid artery (ICA), or in the presence of
a persistent trigeminal artery. Carotid dissection is an increasingly recognized cause
of anterior circulation ischemia. It is possible that in the setting of certain cerebral
hemodynamics, embolism from carotid dissection could cause PCA infarction via a patent
posterior communicating artery. Case Description: We report the MR and MR angiography findings of cerebral dynamics whereby a patient
presents with posterior circulation symptoms from anterior circulation pathology:
left ICA dissection with subsequent left PCA territory infarction. Conclusions: Cerebral hemodynamics may allow PCA infarction from carotid disease even in the
absence of a fetal PCA origin. This report also broadens the spectrum of stroke associated
with carotid dissection.
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References
- Occipital infarction with hemianopsia from carotid occlusive disease.Stroke. 1989; 20: 409-411
- Internal carotid artery dissection in a community. Rochester, Minnesota, 1987–1992.Stroke. 1993; 24: 1678-1680
- Central retinal artery occlusion from carotid dissection diagnosed by cervical computed tomography.Stroke. 1994; 25: 1271-1272
- Anatomical studies of the circle of Willis in normal brain.Arch Neurol Psychiatry. 1959; 81: 409-418
- Occipital lobe infarction from a carotid artery embolic source.J Neurol Ophthalmol. 1996; 16: 33-35
- Arterial occlusions in the vertebrobasilar system. A study of 44 patients with post-mortem data.Brain. 1973; 96: 133-154
- The circle of Willis—The incidence of developmental abnormalities in normal and infarcted brains.Brain. 1967; 90: 747-758
- Bilateral occipital infarctions associated with carotid stenosis in a patient with persistent trigeminal artery.Stroke. 1994; 25: 1520-1523
- Occurrence and mechanisms of occlusion of the anterior cerebral artery.Stroke. 1983; 14: 952-959
- Platelet emboli in rat brain cross when the contralateral carotid artery is occluded.Stroke. 1991; 22: 1053-1058
- Cranial nerve palsy in spontaneous dissection of the extracranial internal carotid artery.Neurology. 1996; 46: 356-359
- Dissections of cervical and cerebral arteries.Neurol Clin. 1983; 1: 155-182
- Headache and neck pain in spontaneous internal carotid and vertebral artery dissections.Neurology. 1995; 45: 1517-1522
- Spontaneous carotid dissection with acute stroke.Arch Neurol. 1987; 44: 137-140
- Spontaneous dissecting aneurysms of the cervical internal carotid artery.Br Med J. 1964; 2: 790-792
- Valve strands are strongly associated with systemic embolization: A transesophageal echocardiographic study.J Am Coll Cardiol. 1995; 26: 1709-1712
- Mitral valve strands in patients with focal cerebral ischemia.Stroke. 1996; 27: 1183-1186
- Enhanced detection of intracardiac sources of cerebral emboli by transesophageal echocardiography.Stroke. 1991; 22: 734-739
- Papillare exkreszenzen an der semilunarklappe der aorta.Wien Med Wochenscher. 1856; 6: 244-247
Article info
Publication history
Accepted:
August 1,
1997
Received:
March 4,
1997
Identification
Copyright
© 1998 National Stroke Association. Published by Elsevier Inc.